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The senescent levels of PDL3 and PDL9 chondrocytes were assessed by senescence-associated beta-galactosidase (SA--gal) staining and qRT-PCR analysis of senescence-associated marker genes

This signaling axis is central to BPC-157s ability to restore endothelial function following insult/damage to the human body

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Dysfunction at the receptor and post-receptor level, rather than insulin deficiency alone, drives disease progression