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Most current therapeutic candidates for NDDs face challenges in crossing the bloodbrain barrier, substantially limiting their clinical efficacy (Ross and Poirier, 2004)
In summary, the accumulation of TMAO will not only increase oxidative stress and endothelial cell damage and permeability but also lead to an increase endothelial-derived NOS expression (56), thereby aggravating disease progression
The peptide could be degraded, the water could be contaminated, or the vial temperature may have caused precipitation
It was shown that targeting Nedd4-2 would promote cellular EnaC expression and exacerbate Liddle syndrome[64]
The association between SSRIs and PSQI scores, as well as its sub-components, was assessed, with non-users of any antidepressant serving as the reference group
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