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Decreasing the liberation of glutamate from the glutathione cycle leads to decreased cortical neuron glutamate, while decreasing the utilization of glutamate increases total glutamate by about 25%

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Experimental work in isolated vascular tissue and cell culture models indicates interactions involving regulators such as endothelial nitric oxide synthase, Src kinase signaling, and vascular endothelial growth factor receptor pathways

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