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Figure 1 2 MASLDs pathophysiology and current treatment MASLD is characterized by an abnormal accumulation of lipids in hepatocytes, commonly occurring in the context of obesity, insulin resistance, and T2DM-key features of metabolic syndrome that substantially affect patient quality of life and long-term prognosis ( The complex pathophysiology of MASLD calls for integrated, multi-faceted therapeutic regimens and improved diagnostic tools to enhance clinical outcomes

NEAAs synthesized from glutamine Although glutamine has been considered an NEAA that is synthesized endogenously, most cancer cells cannot proliferate or survive in a medium that does not contain glutamine 5

(PubMed) Ashton E, Windebank E, Skiba M, et al

Hepatic metabolic adaptation in a murine model of glutathione deficiency

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