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[8] This type of injury does not have a clear dose-response nor temporal relationship, and most often does not have predictive models

Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage

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Wirth, T

When SETD1B is functional, mycolactone exposure leads to the upregulation of CHAC1, resulting in decreased GSH levels and increased oxidative stress, ultimately causing apoptosis

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