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Semaglutide weight loss mechanism #1: it prevents blood sugar spikes Well-functioning L-cells located in the gut produce natural GLP-1 after food intake, which inhibits potentially damaging postprandial glucose excursions (PPGEs) in laymens terms, post-meal blood sugar spikes (PPG inhibition is why, before the FDA approved GLP-1 analog semaglutide for weight loss therapy, it was commonly used in the treatment of diabetes.) This blood sugar-limiting effect mediated by a complex interplay of hormones is called glucose homeostasis. Semaglutide, a GLP-1 analog, does the same thing to regulate glucose homeostasis, but with greater efficacy due to the concentrated dosage used in therapy
f , Change in fat mass
Risk of pancreatic cancer associated with GLP-1 receptor agonist therapy
An analysis of electronic health records from over one million people with type 2 diabetes and no prior diagnosis of dementia found that semaglutide use was associated with a 40 to 70% reduced risk developing dementia over three years, depending on the comparator medication, with the greatest benefit seen when compared to insulin
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