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Nonetheless, our study demonstrates that a persistent imbalance between oxidative stress toxicity and reduced ANTIOX is a key component of Long COVID and that increased aldehyde, neutrophil-associated MPO, protein carbonyl, and NO production are the major neurotoxic pathways, which, when combined with decreased ANTIOX, may have a variety of detrimental effects as reviewed before [81]

Bibcode:2015PLoSO..1019479L

used APEX2 technology to explore the neighboring proteins of the copper chaperone Atox1 and conducted mass spectrometry analysis to identify a new nuclear copper-binding protein called cysteine-rich protein 2 (CRIP2), which interacts with Atox1 in the cell nucleus

Although a clear causal relationship has not yet been proven, reports of liraglutide-associated cholelithiasis and pancreatitis warrant attention

Effect of naloxone on ischemic acute kidney injury in the mouse

doi: 10.1016/S0021-9258(18)77062-2