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Bouyeh M, Gevorgyan O
We thus explain the enzymatic inactivity of ROXY9 by a poor scaffold site for standard glutathionylated substrates
In sickle cell anemia oxidative stress can trigger eryptosis and the release of red blood cell microparticles (RBC-MPs), which are small phospholipid-containing vesicles implicated in physiological processes such as hemostasis and potentially linked to macrovascular dysfunction
Simultaneous analysis of the majority of low-molecular-weight, redox-active compounds from mitochondria
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Nf2, nuclear factor erythroid 2-related factor
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