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Semaglutide activates GLP1 receptors, improving incretin function and insulin secretion (glucose-dependent), inhibiting glucagon release and suppressing hepatic gluconeogenesis, resulting in reductions in both fasting as well as postprandial glucose [10]

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Treatment is less likely to cause serious side effects when done by professionals

GLP-1 medications work by mimicking the natural hormone glucagon-like peptide-1 , which regulates appetite and blood sugar

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