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It is caused by repeated amplification of the CAG trinucleotide in the huntingtin (Htt) gene on the short arm of chromosome 4, which leads to a prolonged polyglutamine stretch at the N-terminus of the protein

Though controversial in longevity medicine, its systemic regenerative effects are well-documented in deficient populations

Studies have demonstrated that miRNAs regulate ferroptosis through direct and indirect approaches

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PLoS ONE 10 , e0122124 (2015)