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Ongoing research is exploring new formulations, such as oral semaglutide, and dual agonists such as tirzepatide
Our data suggest that dulaglutide raised force of contraction in the isolated paced human atrium via GLP-1R involving cAMP-dependent protein kinase in HAP
The GLP-1 component does what researchers already know GLP-1 agonists do
-- Source: [1] JCI (2025): [2] Kidney Int (2024): [3] AJPRP (2015): [4] Abstracts: Tim Fitzpatrick, use of GLP-1 in kidney diseases is a very appealing topic
Theres likely to be innovation against these, but we suspect that theyll remain a barrier
however, these benefits are compromised by PPAR-mediated side effects, primarily weight gain
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