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These models demonstrate the distinct temporal and pathological progression of AD ike features, including tau abnormalities, intraneuronal A accumulation, and synaptic and neuronal loss, making them valuable tools for studying disease mechanisms and therapeutic strategies
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Further clinical studies are warranted to validate its long-term safety, optimal dosing, and therapeutic efficacy in human subjects
These include vitamins (C, E, and -carotene), some metallo-enzymes including glutathione peroxidase (GPx, containing selenium), catalase (CAT, containing iron), superoxide dismutase (SOD, containing copper, manganese and zinc)
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