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Nicotinamide adenine dinucleotide (NADH) and acetyl-CoA, two metabolic intermediates produced in the course of FAO, induce activation of PDK, which in turn phospho-inactivates the E1 subunit of the PDH complex, leading to lower rates of glucose oxidation and higher rates of lactate release 70

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2008 May 15;453(7193):314-21

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The injection site becomes abscessed (hot, hard, red, painful)

In radiation therapy, while SLC7A11 expression is suppressed, ACSL4 is enhanced, potentially exacerbating lipid peroxidation damage [150]