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The poly dopamine shell responds to a high ROS environment to release rutin, directly scavenging excess ROS within mitoROS, repairing disrupted ETC function, and stabilizing MMP, blocking abnormal opening of the mPTP and ferroptosis cascades, ultimately alleviating damage to renal tubular epithelial cells, significantly reducing serum LDH leakage, improving glomerular filtration function and histopathological damage, and achieving protective effects on IRI kidney function [57]

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For instance, a number of variants in the GCL gene have been associated with cystic fibrosis [39], and COPD [40]