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What they found: In the control group 70 % of rats died within 30 days In the BPC-157 group only 10 % died Histology: faster formation of collaterals, normalization of portal vein pressure, 80 % reduction in liver thromboses Why it matters: These are extreme conditions
Downregulates pro-inflammatory genes and those promoting fibrosis
BPC-157 engages the NO system in an interesting way: it does not act in one direction
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Studies in rodent models have shown that BPC-157 appears to upregulate the expression of VEGF (vascular endothelial growth factor) and its receptor VEGFR2, both of which are central to new capillary formation
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