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Moreover, due to their ability to slow gastric emptying, these drugs may compromise the effectiveness of oral medications

Thus, we next sought to examine the role of glycosylation in the stability of proIGF-1Ea

37 In our study, we found that Clopidogrel downregulated the expression of phosphorylated eNOS and induced ER stress in the rat gastric mucosa cells and the BPC 157 protected against Clopidogrel-induced gastric mucosa dysfunction by alleviating ER stress and promoting eNOS -mediated angiogenesis whereas the effect of L-NAME is completely the opposite

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