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In cell and tissue models, GHK-Cu has been associated with upregulation of genes tied to collagen types I and III, elastin, decorin, several matrix metalloproteinases and their inhibitors, and pro-angiogenic factors such as VEGF and FGF2
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2011 in the Journal of Applied Physiology characterized BPC-157's effects on tendon fibroblast outgrowth through increased FAK phosphorylation and downstream paxillin activation, with measurable effects on cell migration rate and tendon-derived fibroblast survival after mechanical injury
But the process can only be as good as the product you start with
however, external factors such as stress and aging cause it to be depleted in our bodies