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The following GSH- or cysteine-delivering strategies are not only useful in the treatment of cigarette smoking-induced lung disease but also are beneficial in general in various inflammatory lung diseases where GSH levels have been shown to be altered (35, 40, 146, 217, 235)

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In the GCH1-BH4 pathway, BH4 functions as an antioxidant capable of trapping peroxidized lipid radicals, with GCH1 serving as the rate-limiting enzyme in BH4 biosynthesis