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a.k.a UMN in mice) of well characterized mouse models of TDP-43 pathology, both display prominent mitochondrial defects in a cell-type specific manner 16

Source: Xie, Y., Choi, T

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This disruption of the physical barrier, coupled with Ang II-mediated impairment of the chemical barrier, creates an imbalance between intestinal epithelial injury and regeneration, setting up a vicious cycle