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PMID 24626261
286 , R967R974 (2004)
Statements Author contributions LD, MT, and JS Collected that data and samples
These findings support the hypothesis that glycocalyx disruption contributes to the pathophysiology of DCI through mechanisms such as neuro-inflammation, microthrombosis, endothelial dysfunction, and oxidative stress (66)
R.1999Expression of UDPglucuronosyltransferases (UGTs) 2B7 and 1A6 in the human brain and identification of 5-hydroxytryptamine as a substrate.3651(May1999),156162,0003-9861 63
For all seven peptides reviewed, the staffers recommended against making them available for compounding
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