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Despite this, the PKC inhibitor bisindolylmaleimide I (Bis) does not affect cAMP augmentation within the D1/GHSR1a dimer, while pertussis toxin, an inhibitor of the G i/o pathway, eliminates cAMP augmentation (Jiang et al., 2006)

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We hypothesize that polymorphisms in genes encoding proteins involved in these reactions could act as modifiers of the cerebral response to ischemia and impact the resultant stroke volume

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